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https://pubmed.ncbi.nlm.nih.gov/21521170/
Mitochondria: the headquarters in ischemia-induced neuronal death Abstract Due to a lack of efficient treatments, searching for novel therapies against acute ischemic stroke represents one of the main fields in neuropharmacology. In this review we summarize and discuss the role of mitochondrial participation in ischemia-induced neuronal death.
https://www.ncbi.nlm.nih.gov/pubmed/21521170
this review we summarize and discuss the role of mitochondrial participation in ischemia-induced neuronal death. Mitochondria are regarded as the main link between cellular stress signals and the execution of programmed death of nerve cells. Since it was described that the release of mitochondrial proteins such as
https://www.researchgate.net/publication/51078019_Mitochondria_the_Headquarters_in_Ischemia-Induced_Neuronal_Death
In this review we summarize and discuss the role of mitochondrial participation in ischemia-induced neuronal death. Mitochondria are regarded as the main link …
https://www.semanticscholar.org/paper/Mitochondria%3A-the-headquarters-in-ischemia-induced-Jord%C3%A1n-Groot/eb4cc3bd56dd74a4521af1b960feaf0e02c9c9e4
Due to a lack of efficient treatments, searching for novel therapies against acute ischemic stroke represents one of the main fields in neuropharmacology. In this review we summarize and discuss the role of mitochondrial participation in ischemia-induced neuronal death. Mitochondria are regarded as the main link between cellular stress signals and the execution of programmed …
https://www.ingentaconnect.com/content/ben/cnsamc/2011/00000011/00000002/art00003
Mitochondria: the Headquarters in Ischemia-Induced Neuronal Death Buy Article: $68.00 + tax (Refund Policy) Authors: Jordan, Joaquin; W.J. de Groot, Piet; F. Galindo, Maria. Source: Central Nervous System Agents in Medicinal Chemistry (Formerly ...
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6147588/
Mitochondria, as the powerhouse of cell, play a critical role in cell energy homeostasis and are thus inevitably involved in the ischemic neuronal death. After ischemia, due to the reduced blood supply, the energy balance is disrupted and adenosine triphosphate (ATP) synthesis is disturbed.
https://pubmed.ncbi.nlm.nih.gov/30271667/
Stroke is the leading cause of death and adult disability worldwide. Mitochondrial dysfunction has been regarded as one of the hallmarks of ischemia/reperfusion (I/R) induced neuronal death. Maintaining the function of mitochondria is crucial in promoting neuron survival and …
https://www.frontiersin.org/articles/10.3389/fnagi.2022.785761/full
Mitochondrial dysfunction has been regarded as one of the major contributors of ischemic neuronal death after stroke. Recently, intercellular mitochondrial transfer between different cell types has been widely studied and suggested as a potential therapeutic approach.
https://www.nature.com/articles/s41419-022-04782-0
Neuronal mitochondrial dynamics are disturbed after ischemic stroke. Optic atrophy 1 (OPA1) and its GTPase activity are involved in maintaining mitochondrial cristae and inner membrane fusion. This...
https://www.semanticscholar.org/paper/Mitochondrial-membrane-potential-and-ischemic-death-Iijima/533d9881ae422bad34aa2c74b3320775c34b5ec5
Mitochondria are intracellular organelles in which high energy phosphate is produced, and it is likely that a significant relationship exists between inactivation of electron transport and apoptosis. Mitochondria are intracellular organelles in which high energy phosphate is produced. Ischemia causes depletion of the materials necessary to produce this phosphate and strongly …
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